HIV and HLA similarity

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Association between presence of HLA-B*5701, HLA-DR7, and HLA-DQ3 and hypersensitivity to HIV-1 reverse-transcriptase inhibitor

Centre for Clinical Immunology and Biomedical Statistics, Royal Perth Hospital and Murdoch University, Perth, Western Australia, Australia (Prof S Mallal FRACP, D Nolan FRACP, C Witt PhD, G Masel MBBS, A M Martin PhD, C Moore BSc, D Sayer Bsc, C Mamotte PhD, D Maxwell, Prof I James PhD); Department of Clinical Immunology and Biochemical Genetics, Royal Perth Hospital (S Mallal, C Witt, G Masel,...

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Role of HLA Adaptation in HIV Evolution

Killing of HIV-infected cells by CD8(+) T-cells imposes strong selection pressure on the virus toward escape. The HLA class I molecules that are successful in mediating some degree of control over the virus are those that tend to present epitopes in conserved regions of the proteome, such as in p24 Gag, in which escape also comes at a significant cost to viral replicative capacity (VRC). In som...

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KIR/HLA Pleiotropism: Protection against Both HIV and Opportunistic Infections

The compound genotype KIR3DS1/HLA-B Bw4-80I, which presumably favors natural killer cell activation, has been implicated in protection against HIV disease. We show that this genotype confers dual protection over the course of HIV disease; early direct containment of HIV viral load, and late specific defense against opportunistic infections, but not AIDS-related malignancies. The double protecti...

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Role of HLA class I (HLA-A, B) and HLA class II (HLA-DRB, DQB) in HIV-1 patients with and without pulmonary tuberculosis.

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Human immunodeficiency virus (HIV-1) infection causes progressive immune deficiency by the targeted depletion of CD4+ T cells. The mechanism by which HIV-1 achieves this is not completely understood, however chronic immune activation is understood to be an essential feature of progressive HIV-1 infection. Although the mechanism by which HIV-1 induces chronic immune activation is also unknown, a...

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ژورنال

عنوان ژورنال: Nature

سال: 1988

ISSN: 0028-0836,1476-4687

DOI: 10.1038/333215c0